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Stasis Dermatitis (CEAP C4a) in Venous Insufficiency - Buffalo Niagara Vein Treatment Center - Dr Karamanoukian
Tuesday, August 20, 2013



Skin changes are the hallmark of chronic venous insufficiency with the underlying cause being venous hypertension or elevated venous pressures in the lower extremities. The end result of chronic venous insufficiency and untreated venous hypertension is advanced trophic changes in the skin with the development of skin breakdown and formation of venous stasis ulcers.


Skin changes related to chronic venous insufficiency include hyperpigmentation, stasis dermatitis, fibrosis of the skin, atrophy of the skin and subcutaneous tissue, and ultimately ulceration known as a venous stasis ulcer.  The incidence of venous stasis ulcer is less than 1% in the general population.  Overall, the skin changes named above are somewhere between one in five individuals with venous insufficiency.


It is well known that venous insufficiency results from a combination of reasons including failure or atrophy of the calf muscles, venous valvular dysfunction, and obstruction to venous blood flow from deep vein thrombosis.


Stasis dermatitis, also known as gravitational eczema or venous stasis eczema can occur in combination with varicose veins, hyperpigmentation of the skin, lipodermatosclerosis, atrophe blanche, and venous ulcerations.  Individuals can also have associated phlebitis, thrombophlebitis, and secondary lymphedema.


Atrophe blanche is whitening or blanching of the skin because the veins and capillaries in the skin have disappeared or have ‘died’.


If you have advanced stages of venous disease including stasis dermatitis, hyperpigmentation of the skin, lipodermatosclerosis, healed venous stasis ulcer, or active venous stasis ulcer, contact Hratch Karamanoukian, MD FACS at the Vein Treatment Center with offices in Williamsville, New York and Clarence, New York.  You can also call to set up an appointment at 716-839-3638 or contact Dr. Karamanoukian via or or are partners with



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